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A great number of cells expressing and co-expressing such markers were detectable in the cerebellum of peripuberal and puberal rabbits, in overt contrast with their complete absence in the postnatal cerebellum of rodents [9], [40], [41].
The cameras were positioned so that all anatomical markers were detectable in the one image.
However, the sympathetic markers were detectable in line N2 and TYRP1 in line N4.
Importantly, none of these neuronal markers were detectable inundifferentiated cells, suggesting the lack of spontaneous neuronal potential.
All five measured brite/beige markers were detectable: the expression of three increased and two remained unchanged from the transition to the white adipose phase.
Consistent with cancer cell propagation, a number of known proliferation markers were detectable, i.e., PCNA, antigen Ki-67, 14-3-3 14-3-3 14-3-3hibitin-PHB [ 15, 19].
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No clear relationship between both markers was detectable.
In contrast to CD68 and CD11c, in the follicles no expression of the M2 markers was detectable.
Cells expressing Ki-67 (a cell cycle protein used as a proliferation marker) were detectable at 2 d after ATP injection, and significantly increased at 3 d, but decreased at 7 d (Figure 3A).
It is of crucial importance that the corresponding marker is detected in the two planes and that a minimum of three corresponding markers are detectable in each segment.
However, no studies have validated whether these MeDIP-identified markers are detectable in maternal plasma, let alone their fetal-specificity in maternal plasma and clinical application.
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