Sentence examples for markers we show from inspiring English sources

Using single nucleotide polymorphism DNA markers we show that mycorrhizas in both forest age classes in both villages showed high levels of genotypic diversity, consistent with a reproductive life history predominated by outcrossing via basidiospore dispersal.

Using genetic interaction and molecular markers, we show that most of these phenotypes are caused by increased activity of the Drosophila EGFR.

Using immunocytochemistry for axon-specific markers, we show that diverse axons from brainstem neuromodulatory centers are found in MLH.

By assessing the individual markers, we show that Lrig1 and Plet1/MTS24 are expressed in the same location.

Using nine microsatellite markers, we show that the population is genetically impoverished compared to nearby populations in Sweden, but less so than monitoring programs suggested.

Using microsatellite markers, we show the evolution of a new species of brown macroalga (Fucus radicans) in the Baltic Sea in the last 400 years, well after the formation of this brackish water body ~8 10 thousand years ago.

By assessing tumorsphere formation and expression of stem cell markers, we showed this to be the case in A549 cells, which harbor a Ras mutation, and in two other non-small-cell lung cancer cell lines, H1975 and H1650, driven by activating EGFR mutations.

Even though demography can be a confounding factor when assessing the adaptive value of highly differentiated or height covarying markers, we showed that it is very unlikely that demographic processes alone could create haplotypes like the ones observed at the extremes of the spatial autocorrelation distributions.

In the present study, using a complementary approach by both inhibition and induction of autophagy as verified using the LC3-II marker, we show that autophagy has strong inhibitory effects on IL-1β production.

Using a co-dominant genotypic PCR marker we show for the first time that, in sugar beet, the GA and B-gene pathways are independent for bolt initiation.

Originally discovered as a protein overexpressed in epithelial cancers, and therefore used as a surface tumour marker, we show that EpCAM can have different faces at the extracellular site.