With 63 nuclear markers, we expect 3 to 4 markers per pond to have P-values less than 0.05 by chance alone.
Similarly, in analyses adding in HPA-axis and inflammatory markers we expect that AUC, CAR, and inflammatory changes will mediate the effects of chronic stress on intraindividual cognitive change.
For X-chromosome markers we expect CN = 1, so if the CN was below the cut-off threshold and we inferred CN = 1, we called it a true positive; if it was above and we inferred CN = 2, we called it a false negative.
As a potential diagnostic marker, we expected a lower accuracy than observed in schizophrenia [8], which would encompass a distributed network including the anterior cingulate and prefrontal regions, hippocampus, and basal ganglia.
For each marker, we expect only a small number of cells to have that marker present during synchronization recovery.
18 19 We had insufficient tissue to exhaustively characterise this cell population and selected markers which we expected would best indicate a number of cell types including DC.
Since oJIA had lower laboratory inflammation markers than pJIA, we expected that osteoblastogenesis would be negatively regulated by inflammatory processes.
In the case of the Ensatina data with 46 putative hybrids and three markers, we might expect 5.75 false F1's and would have wanted 10 markers to keep the error rate near 5%.
In our simulation, if instead of using 95% of the QTL in linkage disequilibrium, we had used 100% of the QTL in linkage disequilibrium with markers, we would expect the PSI and GSI results to be practically equal under the assumption of a very large number of markers.
Nevertheless, by using CD134 as marker for targeting, we expected to affect all recently activated auto-aggressive CD4+ T cells present at the time of targeting, including arthritogenic T cells with a different specificity from that of Mt HSP60176 190.
