Sentence examples for markers we analyzed from inspiring English sources
Using antibodies to basal cell markers, we analyzed biopsies with MGH to profile the distribution and evolution of reserve cells and their relationship to these epithelial components.
To assess whether accumulation of pDC in LN of HIV-1 infected patients involved modulation of tissue homing markers, we analyzed the expression of several cell surface receptors on circulating pDC of HIV-1 positive and negative individuals.
The markers we analyzed included: CD62L and CCR7 (lymph nodes); CCR4 (skin); CD18, CD29 (inflamed tissues); CD49d, CD103, CCR9, and integrin β7 (lamina propria and intraepithelial sites of the gut associated lymphoid tissue or GALT).
To investigate whether this was true for other lymphocyte markers, we analyzed an additional cohort of 220 high-grade serous cases from patients known to have macroscopic residual disease following primary cytoreductive surgery.
Using these markers we analyzed for central memory (CD28+ CD95+) and effector memory (CD28− CD95+) subsets as well as naïve population (CD28+ CD95−) within the CD4+ CD8− and CD4− CD8+ T cells and representative FACS analysis patterns for one animal each for the three different groups are shown in Fig. 3A.
To determine whether zebularine-treated hESCs possessed cardiac expression markers, we analyzed the gene expression using RT-PCR.
Using phosphorylated histone H3 (pHH3) as a marker, we analyzed cell proliferation in fetal hearts at E12.5 and E14.5.
We thus gathered a total of 70 conserved markers that we analyzed as a concatenated data set.
The Tag SNP markers that we analyzed were not in high-linkage disequilibrium with one another (Fig. 1).
Using Galt::RFP and RFP KDEL as Golgi and ER markers, respectively, we analyzed salivary glands of third instar larvae at 96 hr AED (a time window that precedes the growth defect phenotype of dGPHR mutants, Fig. 2D) and demonstrate that dGPHR inactivation was associated with severe ER and Golgi disorganization phenotypes (Fig. 6A,B).
In the case of double mutants with wus-1, clv1-1, clv3-2, clv3-2, CLV::GUS marker line, and WUS GUS marker line, we analyzed only plants that also harbored the erecta mutation.
