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A subset of 450 DArT markers was shared with our mapping project [ 18], where Diversity Arrays technology (DArT) genomic libraries were developed from H. chilense accessions.
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The value of DA ij could range from 0 (no shared markers) to 1 (all markers are shared).
Few markers were shared with the GBS dataset.
None of these markers were shared with those detected in the GBS dataset.
In fact, many phenotypic characteristics and molecular markers are shared between these two cell types (cancerous and trophoblastics ones), providing evidences to support this hypothesis.
Due to the fact that a significant number of markers are shared between PGCs and ES cells, the careful study of PGC specification in vitro is difficult.
Since limited number of markers are shared between Affymetrix 550 K (GenADA) and Illumina HumanHap 550/610 platforms (Pfizer and ADNI), we imputed the GenADA data set to the non-singleton HapMap SNPs based on the HapMap III reference haplotypes in unrelated Caucasian individuals.
The molecular genotype of the isolates from both leg and brain biopsies show that the molecular profile based on multilocus sequence (MLS) analysis of both coding (MLST) and more variable noncoding (VNTR) markers is shared with only two clinical isolates (one from Australia, and the other from an organ transplant recipient in San Francisco, California) out of 85 VGI strains typed [10], [32].
Fifty-two of these markers were shared with the Clementine map.
b 256 markers were shared between the two crosses.
Only three markers were shared between the two approaches.
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