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However, with the inclusion of this correction the number of significantly associated markers remained high (2 94).
While MAF and percent of polymorphic markers remained high in the taurine×indicine composite animals and the average MAF for polymorphic markers was 0.24 in the African breeds, fewer markers were polymorphic in the African breeds.
The proportion of skewed markers remained high (46.1% for p < 0.05).
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H4K16ac and H3K36me3 levels at pTERRA are comparable to the more proximal sites, although the levels of both markers remained higher upon senescence (Fig. 5B and D).
Regardless of the data set, i.e., core collection or inferred groups, the average r for linked markers remained higher than for unlinked markers, supporting physical linkage as the main determinant of LD in this core collection.
This was also reflected by the consistency of LD between these populations, since the correlations of r were very high (>0.99) for distances between markers less than 50 kb between the crossbred and the combined BCD populations, and decayed gradually as the distance between markers increased, but still remained high (0.87 to 0.93) for markers that were about 5 Mb apart (see Figure 4).
Four hour pre-stimulation on the other hand, demonstrated substantially reduced transduction, more than two fold lower than 48 h pre-stimulated cells, yet surface expression of primitive markers remained relatively high.
Expression patterns of the α4β1 integrin lung homing markers remained consistently high on CD4 and CD8 T cells isolated from BAL fluid and varied on peripheral T cells.
The correlation gradually decayed as the distance between markers increased but still remained high (0.87 to 0.93), even if SNPs were about 5 Mb apart.
Meanwhile LD values between the two APA associated markers, PV-atct001 and PV-ag004, remained high in the cultivated resistant and wild resistant genotypes with r2 of 0.9163 and 0.5553, respectively.
The expression of the epithelial markers E-cadherin decreased while the mesenchymal markers vimentin and α-smooth muscle actin (α-SMA) increased at 24 h and remained high at 48 h.
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