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To make informed decisions in dosing erythropoiesis-stimulating agents and intravenous iron therapy, clinicians must determine whether differences between current and previous test results for anemia and iron status markers reflect expected variation, a significant change, or an actual trend.
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These causal effects reflect expected results of (ideal) external interventions.
The results of the survival analyses of the individual markers reflect our expected results based on the cellular functions of the respective histone modifications.
Many biological markers reflect the ongoing disease process of RA.
These markers reflect the nature of tryptophan metabolism.
These results were expected for all markers reflecting high cell turnover by a rapid release and a short half-life of several hours such as nucleosomes and CYFRA 21-1 [ 41].
Expression of these markers reflected autophagy levels in burn wounds.
Unfortunately, biochemical markers reflecting cognitive decline are still sparse [ 8].
In recurrent disease, none of the markers reflected prognosis.
Globally, these findings reflect the expected correlation between systemic markers of inflammation and proinflammatory cytokines levels (Table 3).
Considering the result of this study, we expect that sMIC could be a stress marker reflecting disorders of the maternal immune system that impact NK cell angiogenic function at the fetomaternal interface in some women.
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