So far MIP markers have not been widely used in research on non-model organisms.
These tumor markers have not been applied to EUS-guided FNA.
In spite of its prevalence, the biology of fatigue is relatively poorly understood and biological markers have not yet been identified.
A role for complement has been implicated in the pathophysiology, however, elevations of complement activation markers have not been consistently demonstrated in clinical studies.
Animal-specific viruses have previously been suggested as microbial source tracking tools, but specific ovine viral markers have not been reported before now.
However, respiratory prognostic markers have not been adequately evaluated for this population.
However, these two markers have not been validated for blast resistance.
Background: The impact and prognostic value of the redefinition of myocardial infarction (MI) with more sensitive markers have not been evaluated prospectively in a large, less selected population with acute coronary syndrome (ACS).
Furthermore, the effects of sex and ethnicity on the plasma levels of key candidate protein markers have not been fully described.
Although cell surface markers have not been well characterized in these emerging cell types, transcription factors are known to specifically mark cellular lineages [4] [8].
Thus far, efforts to incorporate multiple isotopic markers have achieved only limited success largely because new markers have not always provided complimentary information that increases the resolution for geographic assignment [19], [24], [25]; [but see 23], [26].
