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In the present study, the use of cluster analysis rather than the restrictive definitions of metabolic syndrome used elsewhere to assess associations with inflammatory markers enabled us to identify a broader section of the adolescent population at risk of cardiovascular and metabolic disease.
Nonetheless, the extensive sampling of the genome provided by the RAD markers enabled us to construct a linkage map based on recombinational distances between paternally inherited RAD markers.
The mapped markers enabled us to arrange 265 scaffolds of the Nasonia genome assembly 1.0 on the linkage map, representing 63.6% of the assembled N. vitripennis genome.
Five specific markers enabled us to segregate micro- and macro-follicular adenomas from oncocytic adenomas and minimally invasive follicular carcinomas, while only one of the six cross-validated markers showed the same performance.
Screening with molecular markers enabled us to identify three chromosome regions associated with this phenomenon.
The previously mapped markers enabled us to align and integrate the different maps in the present study.
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The detailed analysis of homoelogous and nonhomoeologous positions for each marker enables us to propose some hypotheses relating to the evolution of loci for resistance to powdery mildew in wheat.
We used graphical genotyping to compare the marker scores (A or B) and the phase (A or B) of the markers, which enabled us to identify whether each marker was present or absent in a particular progeny isolate.
Careful examination of individual markers has enabled us to dissect this relationship.
DArT markers also enabled us to assess genetic diversity among four agricultural Brassica species that are extensively being used in rapeseed improvement programmes.
The female's FRT chromosome contained either a dominant (P{y+}) or recessive (e ) marker that enabled us to select in subsequent generations for animals who did not carry the marker, and hence, carried a mutagenized FRT chromosome.
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