Sentence examples for markers discussed in from inspiring English sources

Simulation of the markers discussed in Section 2.3 was undertaken for each of the models, with a hERG-only or multi-channel block, at low, medium, high, and overdose EFTPCs.

We might therefore assume that the changed RBC shape seen in all the PD patients might be due to the upregulation of the inflammatory markers discussed in the previous paragraphs, and that the iron plays a less prominent role.

The authors of this manuscript have the following competing interests: Austrian Institute of Technology has filed a patent application based on the methylation markers discussed in this article, with MW, AW, KV, RZ and AG as inventors.

However neither the glymphatic circulation proposal nor the convection-assisted movement hypotheses can easily reconcile the observation that the fluorescent markers are preferentially located in perivenous spaces at late times with the data for preferential periarterial location and efflux of other markers discussed in section 4.1.

The markers discussed here have been studied in different disease settings and with different rigor, ranging from meta-analyses of several clinical studies to promising evidence in preclinical studies (Table 7).

Many possible biochemical markers were discussed in our previous review [ 2].

The calculation of such scores can be relatively simple or can be based on complicated formulas (e.g. those underlying Fibrotest/Fibrosure). GGT: γ glutamyl transferase, HA: hyaluronic acid, TIMP-1: Tissue inhibitors of matrix metalloproteinase- 1, ALT: Alanine aminotransferase, AST: Aspartate aminotransferase The most commonly used markers are discussed in details below.

Some opportunities and methodologic issues associated with using markers are discussed in the context of exposures to four complex mixtures: environmental tobacco smoke and nitrogen dioxide, acid aerosols and oxidant outdoor pollution, environmental tobacco smoke and radon, and volatile organic compounds.

We hypothesize that the gene silencing starts in the 35S promoter and then gradually spreads into the NOS promoter of the resistance marker, as discussed in Mishiba et al.[ 63].

For this method, we used Gustavo de los Campos' software written in R. The code implements the calculation of the ridge regression estimation of the marker effects discussed in the ridge regression section and uses these estimates to perform the prediction for the individuals in the testing set.

Recently, Suh et al. [ 49] documented that the downregulation of miR-145 in PCa could play a role in cancer initiation, and that the mechanism for its regulation is mediated by DNA methylation and p53 mutation pathways (a protein marker discussed later in this review).