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However, neither [TIMP-2]·[IGFBP7] (Fig. 2) nor individual markers (data not shown) discriminated well for AKI at the 18-h (pre-resuscitation) time point.
As an alternative, a downstream biological measurement such as the white blood cell count, C reactive protein, and procalcitonin was analyzed, but we did not find any significant relationship of IL-6 rs1800795 polymorphism with these markers (data not shown).
The cultures were negative for dopaminergic (TH), cholinergic (ChAT) and serotoninergic (serotonin) markers (data not shown).
Flotillin-1 and -2 also co-localized with uropod markers (data not shown).
However, we could identify the STK35L1 locus by using some other conserved markers (data not shown).
Whatever the experimental condition (hypoxic and normoxic conditions) we never detected ECs markers (data not shown).
NT2N displayed no staining for undifferentiated progenitor or oligodendrocyte markers (data not shown) but did display staining for all of the neuron markers and GFAP (Fig. 1D F).
The study revealed that the two conserved TBCD protein segments were directed to the centrosome, co-localizing with various centrosomal markers (data not shown).
However, restoring PPARγ expression in null cells only modestly altered labyrinthine marker expression (Figure 11) and did not alter giant cell markers (data not shown).
Staining was not seen with the neuron, astroglial or oligodendrocyte markers (data not shown) with the exception of β-tubulin III (BT3) and GFAP.
Although labelling was observed in essentially the same cell populations, we observed little sub-cellular colocalization of these two markers (data not shown).
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