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Several factors, such as nutrition, nicotine consumption, medication and physical activity should be controlled for when inflammation markers are studied.
Where single markers are studied, reliance remains on good clinical data about ancestry.
Several tumor markers are studied to explain the mechanism of pancreatic carcinogenesis.
It comes as no surprise that species with wide geographical distributions consist of two or more evolutionary units when molecular markers are studied.
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Cell markers were studied at 25 days of culture.
Gene expression and the immunostaining of myogenic and neurogenic markers were studied.
The association between SSTR2 status and molecular markers was studied with Pearson chi-square analysis by cross-tabulations, and difference in Ki67 with independent samples t test.
The performance of these markers is studied based on the following two aspects: the spatial accuracy of the local heat release rate and the trend in the total heat release rate with equivalence ratio.
The segregation data of the remaining markers were studied with the program MultiPoint ([95] [97]; http://www.multiqtl.com).
Alterations in mRNA expression of oxidative stress and apoptosis markers were studied in PC12 cells exposed to MCP (10−5 M) for 6 h.
In a final step the involvement of the type IV DNA secretion systems encoded in the GGI and the conjugative plasmid in the spread of chromosomal markers was studied.
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