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Certainly, processes besides proliferation contribute to breast cancer metastasis, however this does not indicate that proliferation markers are improved by additional genes.
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After initial IV therapy, switching to oral antibiotics is generally accepted when patients are afebrile, inflammatory symptoms are regressing and inflammatory markers are improving [ 16, 17].
Nevertheless, the vitamin K status markers were improved after supplementation.
In contrast, K-877 100 μg BID had little or no effect on CV or renal markers, and hepatic markers were improved.
CT scans/MRI findings as well as tumour markers were improved following induction chemotherapy in seven patients correctly indicating a favourable outcome.
However, although oxidative damage markers were improved by nifedipine treatment, we did not detect significant changes in VEGF or sVEGFR-1 concentrations.
In comparison with the performance of each single marker alone (Fig. 5), these data imply that in combination the sensitivity and specificity of the markers can be improved.
It is therefore likely that the correlation between these two markers could be improved when comparing post-exercise pCort values with sCort sampled with a delay of 10 20 min.
Although we showed that the amplification success of existing microsatellite markers can be improved by redesigning primer sets, heterozygosity was generally lower in test than source species.
It is possible that the sensitivity with this marker can be improved if more PCR cycles are performed.
Moreover, combining MRI with protein measures did not improve predictive power in contrast to previous studies where CSF marker performance was improved in combination with MRI [41].
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