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Cells engineered to detect a cancer-associated marker produce a protein that sensitizes them to an anticancer drug.
In these diploids, crossovers between the centromere and the heterozygous SUP-o marker produce red/white sectored colonies.
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The GS5-indel1 marker produced a 63-bp band (MG allele) and a 67-bp band (WG or NG).
The most specific and discriminatory results were shown using the TP53 gene since this marker produced greatest fragment size differences between animal species studied.
Primers designed from the sequence of the RAPD marker produced a diagnostic amplification product of 346 bp for GV4 alone, distinguishing it from all other test isolates.
The results showed that the digestibility coefficients obtained with total collection and with acid-insoluble ash marker were similar (80.5±2.73% vs. 84.4±5.21%, respectively), whereas using a chromic oxide marker produced a significantly lower value (61.7±2.0%).
For instance, if a marker produces high signals for all individuals but a few, the latter could harbor the fourth type of nucleotide in the extension region.
Our results showed that a stronger LD and a lower MAF for an untyped marker produced better ARs for all the five methods.
A monolayer marker produces more reliable results than staining the LD core with a fluorescent dye, particularly as imaging through the LD in single x y planes allows demonstration of the continuation of the membranes during LD fusion.
Amplification with the primers developed for the CAPS marker produced a single PCR amplicon of the expected 311 bp size.
Therefore, as a unique marker produced by the neutrophil, serum Hp-MMP 9 complexes should herald early changes in neutrophils associated with the host response to inflammation.
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