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As of 2010, assays that incorporate an array of antibodies against specific protein marker molecules are an emerging technology; there are hopes for these multiplex assays that could measure many markers at once.
In the majority of cases, the marker molecules are expressed by the tumour cells themselves or by cells of the tumour microenvironment.
In addition, whether the potential CSC-specific marker molecules are implicated in the development of DNM in OSCC has not been examined.
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However, isolation of bonafide regulatory T-cells remains difficult because the availability of specific marker molecules is still limited.
The expression of marker molecules was analysed by qRT PCR.
However, unlike in the ensemble case, where the position of the bright marker molecules is given by the position of the zero, the exact position of the switched-on or activated molecule is unknown.
Only recently has the surface marker expression of human MSC from different dental tissues been thoroughly investigated and a useful panel of identifying marker molecules been recommended [ 73, 80].
As shown in Table 1, the expression of these two marker molecules was significantly correlated (p = 0.007).> -wrap-foot> *significantly significaccordingding to Fisher's exact test To determine the predictive risk factors of DNM, the association between clinicopathological factors and DNM was examined in the 50 cases of stage I/II TSCC.
However, until an activation-independent surface marker molecule is found for Treg, this question cannot be answered to date.
To date, a number of marker molecules have been found in NSCs, e.g., nestin, Sox2, Musashi-1, SSEA-1 (stage-specific embryonic antigen-1; also known as Lewis x antigen or CD15) and prominin-1 (CD133).
In absence of Lhx2, a number of dorsal-thalamus-specific markers or patterning-related molecules are normally expressed, implying the normal development of dorsal thalamus nucleus.
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