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Treatment of malignant gliomas using genetically-modified neural stem cells as a marker is discussed.
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In this review, the clinical applications of plasma EBV DNA as a tumor marker for NPC and the biological characteristics of this marker are discussed.
Available E. faecium genome data were examined for the presence of IS 16 homologous sequences and a genetic association of IS 16 with genomic markers is discussed.
The use of bone markers is discussed controversially in the literature, and no clear recommendation can be given at the moment [1, 8].
Also, future development of integrative systems of genomic risk markers is discussed.
We also studied the levels of ERα phosphorylated forms whose involvement as prognostic markers is discussed below.
In this paper, design strategies for clinical trials aimed at disclosing predictive markers are discussed.
The implications of these results for the two tasks as markers are discussed, with special focus on those earlier studies that did not evaluate unmedicated patients.
Specific features of several of these markers are discussed below.
The reasons for the discrepancy between the biparental markers and the uniparental markers are discussed.
Many possible biochemical markers were discussed in our previous review [ 2].
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