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Because yeast transformation is performed using plasmids in our system, the marker genes are lost in the absence of selection pressure.
The independently expressed ECFP and mCherry selection marker genes are used as controls to monitor the efficiency of construct transfection and gene expression in the treated cells in the BiFC system.
One reason is the presence of resistance genes in transgenic strains, which have been introduced in the bacteria during the allelic replacement process, and methods avoiding use of such marker genes are therefore highly desirable.
Primers for the mouse marker genes are presented in Table S3.
Transposon marker genes are mini-w+ for f02676 and EYFP for PL00420.
These data infer that flanking marker genes are conserved in micro-syntenic environment corresponding to STK35L1 synteny.
These top-ranked marker genes are valuable because they are quantitatively more overexpressed than the other marker genes and thus increase the sensitivity of cancer diagnosis.
The influences of laminar flow and interstitial flow on expression of SMC marker genes are mediated by activation of ERK1/2 MAPK.
Hematopoietic cells derive from hemogenic endothelial cells, which express Tie2, Flk1, VEC, and endoglin all markers of endothelial cells [26] and expression of these endothelial marker genes are decreased after hematopoietic commitment and differentiation.
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This study has confirmed that a panel of 'marker' genes are required to identify tendon cell phenotype from other mesenchymal tissues.
In the OVA model, certain groups of genes, also called the 'marker genes' are only over-expressed (or under-expressed) in samples belonging to a particular class and never in samples of other classes.
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