Sentence examples for marker for point from inspiring English sources

Given that many authors use p53 overexpression as a marker for point mutation this issue is of some importance.

A general trend in all these studies is that COI is an effective marker for pointing at host- and eco-specific taxa in allegedly morphologically undistinguishable groups.

Multiple mutants which we constructed were confirmed by genotyping, using wild-type and T-DNA allele-specific PCR for insertional alleles, and CAPS [ 124] or dCAPS [ 125] markers for point mutation alleles; except for max4-1, whomozygosityosity was confirmed by testing progeny for uniform BASTA-resistance.

Delivery of red elemental nanoparticles for the antibacterial study in P. aeroginosa strain JS-11 showed that it could serve as molecular marker for end point, qualitative, and quantitative antibacterial assays [73].

After validation by sequencing of the mutations within saposin C, the loxP site was used as a marker for the point mutation genotyping with the primers C loxP F (5′-CAGATACCCCATCCTCCCATGG-3′) and C loxP R (5′-TGTGAAGGACAAGAGGAGAGTG-3′).

In these instances, the more reliable markers for stage 83 (sugar inflection point) and stage 87 (start of rapid flesh softening) should be used (Table 1).

AS he steers his snub-nosed truck through Midtown traffic, up to 1,000 gallons of used restaurant grease sloshing in a tank in the back, Axel Arce keeps an eye out for sights that can prompt a bit of New York trivia, like the fact that the statue at Columbus Circle is the traditional distance marker for New York, the point from which mileage between this city and all others are measured.

For a better visualization (due to the high density of the obtained points), we have used one marker for each 30 points in the curve of the proposed approach.

The association between markers of fibrin degradation in patients with progressive breast cancer suggests that the D-dimer level is a clinically important marker for progression and points towards a relation between haemostasis and tumour progression.

Our review of the growing literature on early markers for autism points to a distributed impairment as a likely cause for this disorder.

Metabolic profile changes reported herein are followed by the appearance of molecular markers for apoptosis, pointing to the fact that if tumour cells experience interruption of the altered metabolic network that turns glycogen rapidly into nucleic acid ribose and fatty acids, that interruption will impose a blockage on cell cycle progression.