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Surface marker expression levels were expressed as the geometrical mean fluorescence intensity on monocytes.
The effects of cellular labeling by virus infection in the immune cells confirm the cell proliferation and phenotype marker expression levels in parental DCs and DC/NF cells.
Addition of bovine IL-10 alone did not affect any of the analyzed surface marker expression levels (data not shown).
Statistical analysis of the dose-dependent modulation of DC surface marker expression levels by bovine IL-10 was performed using a one by one correlation model: surface marker expression level or cytokine concentration = bovine IL-10 concentration.
The association between molecular marker expression levels and radiological response is summarized graphically in Fig. 4.
Fig. 4 showed that as mean molecular marker expression levels increase, radiological response becomes poorer.
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Analysis of lung adenoma cells in the absence or presence of Bmi1 did not reveal differences at the morphological and at cell marker expression level.
Neither site of biopsy within the oral cavity, nor the nominal time of biopsy had any significant impact on any of the four markers expression levels.
To evaluate the pathogenetic and prognostic roles of five candidate molecular markers (expression levels of survivin, p53, and pERK1-2 proteins; amplification of CyclinD1 and h-prune genes), we have examined fifty-three patients with diagnosis of T4 breast carcinoma T4-N0-2-M0 T4-N0-2-M0 T4-N0-2-M0 T4-N0-2-M0accordingSobin etoal. [ 1]).
The morphology, marker gene expression levels and stability for long-term culture of these cells were similar to those of fibroblast-derived Ff-hiPSCs (Figures 2 and S3D).
We also detected several ER-stress marker protein expression levels by Western bloting.
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