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Both haematoxylin-eosin analysis and CD45 marker expression are in agreement with these findings.
These vascular defects are not secondary to primary neural defects, as neural morphology and marker expression are normal even subsequent to the onset of vascular pathology.
Quantitative results of the antigenic marker expression are summarized in Fig. 2A.
Cancer cell subpopulations based only upon marker expression are likely to be inaccurate due to antigenic plasticity and therefore studies based upon functional assays are likely more reliable to identify tumor stem cells.
Patient characteristics and tumor marker expression are described in Table 1.
Details of the methods used to determine tumour marker expression are described by Lakhani and colleagues [ 10, 11].
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Double-knockdown hESCs (ShD1D2, ShD2D3, and ShD1D3-hESCs) showed a propensity for spontaneous differentiation into cells expressing endoderm markers, whereas pluripotency and neuroectoderm marker expression was systematically diminished.
CD68 marker expression was found in scattered subepicardial cells, and some of them co-expressed the Lyve1 antigen (Fig. 9f j).
The induction of stress marker expression was assessed by fluorescence microscopy after 48 h.
FSK effects towards BC marker expression were next studied through immunolocalization.
Cell shape features, surface design parameters, and osteogenic marker expression were strongly correlated in vitro.
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