Because the gene expression of deltaC during development can be regulated by a genetic loop of notch pathway self-activation (Holley et al, 2002 [Her1 and the Notch Pathway Function within the Oscillator Mechanism that Regulates Zebrafish Somitogenesis, Development 129 1175-83]), we viewed this rescue of deltaC expression as a marker for restored notch circuitry in mib by zangtl2 overexpression.
Neurexin I and the neuroligins may thus provide important functional markers for analysing neural circuitry in the bee brain.
Finally, we found that the transition in CSPG expression in the striatum, a potential marker for the maturation of circuitry underlying both exploratory and motor behaviours, is also significantly accelerated in enriched mice.
The VBM measures associated with RAVLT scores seem to be a subset of those disease-sensitive markers, showing a specific memory circuitry contributing to the disease, as well as suggesting that the disease is implicated by not only this memory function but also other complicated factors.
Since these systems do not require selection markers or DNA replication to maintain circuitry constructs, there are no limitations on DNA circularization or on plasmid origin of replication and antibiotic compatibility.
To determine whether transplanted iPS cell-derived photoreceptors integrate within the host retinal circuitry, immunocytochemical analysis of synaptic marker expression was performed.
In particular, it is not known if interneurons expressing different chemical markers execute different functions in the CA1 circuitry.
The authors believe that segregation of cognitive and emotional circuitry in at-risk youth may be a marker of weaker cognitive control in FHP adolescents when they are in emotionally laden situations.
The fact that neural overactivity during working memory persists in depressed patients in clinical remission (12) suggests that abnormalities in the neural circuitry supporting working memory might be a vulnerability marker of major depression.
To deeply investigate the effect of PEA on neuronal circuitry, we decided to perform analysis on a neuronal marker, MAP-2.
In the present study, we aimed for the first time to investigate in mice whether genetically determined differences in anxiety-related behavior as well as risk assessment would be reflected by differential stress-induced c-Fos expression as a reliable marker of neuronal activation in key brain areas of anxiety circuitries previously described in rats (for review, see [14]).
