Sentence examples for marker and increased from inspiring English sources

As shown in Fig. 4a,b, bleomycin (5 mg/kg) seemed to induce myofibroblast activation, which was associated with decreased expression of E-cadherin (epithelial marker) and increased expression of α-smooth muscle actin (SMA) (mesenchymal marker).

SAA is a non-specific inflammatory marker and increased concentrations are seen in bacterial infections [ 3, 8- 10], viral infections [ 1, 11] and in sterile inflammation [ 6].

Under neuron differentiation culture condition in both 3A6-hMSCs and primary hMSCs, downregulation of EZH2 decreased CD105 (MSC marker) and increased MAP2 (neuron marker) protein expression (Fig 1C).

With Cd-MEC co-culture, SCs showed a marked and widespread increase in VIM protein, a mesenchymal cell marker, and increased VIM transcript.

Fecal calprotectin is a very sensitive marker for intestinal inflammation, but it is not a specific marker and increased levels are also found in neoplasia, infections, polyps, and with use of non-steroidal anti-inflammatory drugs and increasing age [ 23].

To get some idea of the distance between the transposon-encoded Tetr marker and the beneficial mutation, we examined the co-transduction of the resistance marker and increased cell size, using REL10247 as the donor and the ancestor as the recipient.

In conclusion, environmental lead exposure, monitored by blood lead levels, affects inflammatory marker and increasing of both markers may lead to adverse change in blood pressure.

The integrated map also provides more choice in the type of marker and increases the probability of polymorphic markers in important chromosomal intervals and greater genome coverage than single crosses.

Conversely, overexpression of MLK4 in low-MLK4 cell lines (MCF10A and BT474) resulted in the upregulation of mesenchymal markers and increased phosphorylation of NF-κB (Fig. 6f, g and Supplementary Fig. 12).

hiPSC-CMs harboring the R9C PLN mutation showed activation of a hypertrophic phenotype, as evidenced by expression of hypertrophic markers and increased cell size and capacitance of cardiomyocytes.

Finally, analysis of brain injury indicated that lopinavir/ritonavir treatment resulted in cerebrovascular injury associated with decreased synaptic markers and increased inflammation, and that the cerebral cortex was more vulnerable than the cerebellum or hippocampus.