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All the candidate limbs are marked in Table 3.
Because these 'spikes' can alter the overall peak area integrations, giving false impressions of the total spectral intensity, they are excluded from the integration regions as marked in Table 2.
ERA genes that had differential expression consistent with enriched/depleted cytobands and are involved in cell cycle and/or cell proliferation are marked in Table 2. Dysregulation of these genes may contribute to the differences in proliferation rates between ER+ and ER− cancers.
To distinguish the direct and indirect effects of E2 we added datasets of direct ER targets derived by chromatin immunoprecipitation based studies, and datasets containing both direct and indirect targets characterized by early response to E2 (Text S2.C, with genes within these datasets marked in Table S1).
Pollutants affecting the levels of species reported within this study have been marked in Table 3.
Results from these overlapping cases are clearly marked in Table S4 in the data supplement.
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In the EU ring trials, either extracted DNA or flour samples of reference material were distributed and therefore only four of the in-house validations could be compared to ring trial results (marked in yellow in Table 10).
The final eight sets are marked in Supplementary Table S2.
Significant coefficients are marked in the table with * or **.
Significant differences are marked in the table with **.
The lags that had significant regression slopes in the GLM model are marked in this table.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com