Although studies demonstrated that NG2 glia can differentiate into oligodendrocytes, neurons and astrocytes [9] [12], genetic fate mapping studies did not yet give rise to consistent conclusions about the neuronal and astrocytic fates of NG2 cells in vivo (see [1] for the review).
Finally, low genome coverage of molecular markers in many mapping studies does not allow a genome-wide detection of QTLs with a high resolution, resulting in different QTLs identified from different genetic populations evaluated under different environmental and screening conditions.
While our QTL mapping study does not provide definitive proof that LAR1 controls the variation in the concentration of flavanols in the fruit cortex and skin, recent functional evidence using gene silencing of MdANS strengthens this hypothesis.
Parallel to how example study reduces cognitive load compared to problem solving, learners engaged in concept map study do not have to invest effort in searching for the answer to a question or the connection between concepts, but rather, devote all available working memory resources to studying the connections between concepts.
However, epitope mapping studies alone do not identify the antibodies that are inhibitory and/or pathogenic.
Our localization studies do not map precisely the position of MEI-S332 with respect to the centromere, kinetochore, or centric heterochromatin in anaphase II cells, although they clearly show it is not present along the arms unless at very low levels.
In summary, we are not suggesting that microsatellites should be abandoned in mapping studies, but we do urge that appropriate error checking precautions are employed.
This observation is in contrast with human studies where noticeable differences in breath composition between males and females were reported, although those studies did not comprehensively map the breath metabolome.
These studies did not find a map for numerosity, though scientists have long suspected one exists.
