Suggestions(1)
Exact(4)
Genetic mapping studies confirmed that the progenitor A and C genomes are essentially intact in natural lines of B. napus and have not been substantially rearranged [ 13].
Quantitative trait loci (QTL) mapping studies confirmed the influence of genes other than Prnp in determining incubation time and additional fine mapping and differential expression studies have suggested numerous candidate genes (7– 11).
Although QTL mapping studies confirmed the polygenic control of resistance as e.g. in pea [ 35], faba bean [ 36] and chickpea [ 37], no markers suitable for marker assisted selection (MAS) are available yet.
These studies demonstrated that GC tumor susceptibility is a polygenic, heritable trait involving several Granulosa cell tumor (Gct) susceptibilit y loci on the autosomes and Chr X., Both F2 intercross and recombinant inbred strain mapping studies confirmed the SWR allele at the Gct1 locus on Chr 4 is both necessary for GC tumor development and sensitive to dehydroepiandrosterone (DHEA).
Similar(56)
Hectd2 was first identified in mouse mapping studies and confirmed as a candidate in human prion disease association studies (15).
Genetic fate mapping studies have confirmed that differentiated resting astrocytes can proliferate following injury with a subset expressing nestin (Buffo et al., 2008), supporting a capacity for astroglia to de-differentiate.
Further studies confirmed this.
(Twoother studies confirmed these results).
Further pathological studies confirmed NBTE.
Subsequent studies confirmed these adverse effects.
Later studies confirmed many of his findings.
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