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Furthermore, our previous fate mapping experiments indicated that in wild-type (WT) mice CD8αα IEL with TCRαβ are derived from precursors expressing RORγt, which is an isoform of an orphan nuclear receptor, RORγ, encoded by the Rorc gene [6].
Promoter mapping experiments indicated that each fadD had an independent transcriptional start site, suggesting that they were independently transcribed; however, northern blot analyses indicated that fadD2 and fadD1 can be co-transcribed on a single larger transcript or as smaller independent transcripts (Fig. 3A and 3B).
Genetic mapping experiments indicated that Mt-NS1 is not likely a typical R gene.
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Domain-mapping experiments indicated that the mutant carrying the N terminal CARD domain (1 218) and the mutant carrying the DexD/H-box helicase domain (219 925) interacted with REUL individually (Fig. 2E).
Epitope-mapping experiments indicated that among the subjects who demonstrated post-exposure antibodies to romiplostim, 70% were directed toward the TMP peptide, the active site of the molecule.
Finally, recent genome-scale chromatin immunoprecipitation and DNase hypersensitivity mapping experiments have indicated that DNA-protein interactions evolve rapidly between species.
These experiments indicated rapid activation of Rho and sustained activation of p38 MAP kinase by sVCAM-1 in adult brain microvascular endothelial cells.
Coimmunoprecipitation experiments indicated AGE-LDL interacts with TLR4, RAGE, and CD36.
Dynamic light-scattering experiments indicated the protein to be monodisperse.
Expression experiments indicated successful cloning.
These experiments indicated that a 5- to 6-week interval between the ChAdV63.HIVconsv prime and MVA.HIVconsv boost should be used for eliciting high frequencies of T-cell responses, which will in turn allow detailed mapping of subdominant epitopes.
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