Many other SLN mapping experiments have also been reported in mice [137, 138] and rats [139 141].
Extensive domain mapping experiments have recently confirmed that the N-terminal helical-domain is required for the fusogenic, lytic and surface activities of SP-B [38].
Although previous fate mapping experiments have identified the majority of derivatives of neural crest cells and cranial mesoderm in the jaw, the spatial relationships and the interactions over time between both cell types are not completely understood.
Recent, grafting and fate mapping experiments have unequivocally shown that molecular signals deriving from the endodermal and ectodermal epithelial linings of the 1st PA, are essential for patterning the underlying neural crest-derived ectomesenchyme [29], [30].
Mass spectrometry mapping experiments have helped us understand how Ube2W mediates TRIM5α polyUb "anchoring" to TRIM5α.
Our mapping experiments have also revealed one case of a misassembled contig on chromosome 3.
Recent genetic fate-mapping experiments have suggested that, on the population level, progenitors expressing cut-like homeobox 2 (CUX2), another marker specific for callosal and other superficial-layer neurons, exclusively produce superficial-layer neurons (Franco et al., 2012), although conflicting results were subsequently reported (Guo et al., 2013).
Recent data from live-imaging studies and long-term fate-mapping experiments have demonstrated regionally restricted contributions from multiple distinct stem cell niches in the PSU during homeostasis (Ghazizadeh and Taichman, 2001; Morris et al., 2004; Levy et al., 2005; Jaks et al., 2008; Brownell et al., 2011; Page et al., 2013).
Fate-mapping experiments have shown that the cleavage patterns, as well as the early fate maps of direct and indirect developing hemichordates, are similar to those of indirect-developing echinoids (Colwin and Colwin, 1951; Cameron et al., 1987; Cameron et al., 1989; Cameron and Davidson, 1991; Henry et al., 2001).
Fate-mapping experiments have shown that the liver arises from lateral domains of endoderm in the developing ventral foregut as well as from endodermal cells that track along the ventral midline [ 1, 2].
However, fate-mapping experiments have since established that these cells are restricted to lower hair follicle lineages during steady-state homeostasis (Morris et al., 2004; Jaks et al., 2008; Youssef et al., 2010; Page et al., 2013).
