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To identify the molecular lesion causing the kar mutant phenotype, we mapped the mutation using microsatellite markers.
KS, HN, and SM screened A. thaliana tagging lines, isolated br04, mapped the mutation and analyzed the mutant.
We mapped the mutation to a narrow region, 21B5-21B8, as coconut is fully lethal over several overlapping deficiencies.
To identify the gene affected in the tra mutant, we mapped the mutation to an interval on linkage group 20 flanked by the markers z13672 and z536.
Complementation crosses with ap blot over a set of overlapping ap deletions mapped the mutation to am ∼11-kb interval upstream of ap MM.
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We mapped the mutations on each branch of the tree.
Further DNA sequencing analysis mapped the mutations to 169 pneumococcal genes.
We mapped the mutations to the coding exons of the human genes in the GEnsembl set.
We next mapped the mutations of Pro3-1 on a homology structure model.
We mapped the mutations onto the crystal structure of the PP2A holoenzyme complex to predict their molecular and functional consequences.
We mapped the mutations on chromosome 3 using the markers ru, h, th, cu, sr, e s, and Pr.
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