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In 1911, the first genetic map was achieved by mapping the fruit fly genes.
Importantly, this map was achieved for orders of magnitude less expense than other genome projects.
The target MAP was achieved in most cases with no difference between the groups (Fig. 4).
Overall in the studies, the increase in MAP was achieved by a 1.7 ± 0.4 μg/kg per minute increase in norepinephrine infusion.
In iNOS knockout mice, the target MAP was achieved with less than 20% (P < 0.001) of the NA infusion rate required in wild-type mice.
Gene mapping of bvORF17, bvORF20, and bvORF22 on the physical map was achieved by DNA gel blot analysis, and recombinant phage no. 10 was identified as containing all mapped genes.
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Chemical mapping was achieved on ZnO and ZnWO4 - ZnO samples.
Here, temperature mapping was achieved by using specifically designed thermometers filled with ethylene glycol.
Forty-three of the 51 phenotypes were mapped directly to mammalian equivalents based on MeSH terms searches, whereas for eight phenotypes mapping was achieved after manual searches in the Mammalian Phenotype Browser and inferences drawn on related pathophysiology.
Fine mapping was achieved by backcrossing Mariusz mice to C3H/HeH, C3H/HeN and, finally, to C57BL/6J mice and examining mice with the Mariusz phenotype (low grip strength and fused toes) with a panel of polymorphic markers spanning the region.
Mapping was achieved across array platforms using NCBI Entrez gene ids.
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