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In a mixed-effects linear regression model, change in microvascular reactivity over the first 2 to 4 days of treatment correlated significantly with increasing MAP and decreasing C-reactive protein, but not with change in plasma L-arginine.
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We observed that intravenous administration of JM-1232 decreased MAP and decreased HR in rabbits.
Administration of vasopressin to septic animals (group SV) increased MAP and decreased CI and HR.
Twelve hours after discontinuation of the therapy, transient increases in MAP and decrease in pO2 were observed.
This study examined the relation between MAP and decreased GFR in participants aged ≥ 35 years from the Liaoning province of China.
After a compensatory increase of MAP and decrease of HR during the initial reperfusion stage, the MAP, HR changed gradually toward the value recorded at baseline after the reperfusion in the LT recipients (Table 1).
In a clinical study of septic shock patients, Broccard and coworkers [ 118] reported that nonspecific NOS inhibition increased MAP and decreased cardiac output, allowed gradual withdrawl of α-adrenergic support, decreased DO2 and increased O2ER, but had no effect on VO2, lactate, or splanchnic oxygenation.
With respect to the Niehuis et al. (2010) map, the current map presented here expands the number of mapped scaffolds, increases the percentage of assembled genome that is mapped, and decreases average distance between clusters (see Table 3 for comparisons between the new and previous map).
McMillan et al. [27] found increased axial and radial diffusivity on ADC maps and decreased FA of muscle in dystrophic wild-type mice vs. normal controls.
Addition of markers to the genetic map will likely increase map length and decrease the latter estimate.
Corticosteroids are associated with increased detection of intestinal MAP DNA and decreased expression of certain MMPs.
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