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Many such interactions that may occur between QTL x QTL and QTL x genetic background are unknown.
Recent developments in experimental techniques have uncovered many such interactions and shed new lights on their specificity.
Many such interactions have been discussed so far.
However, many such interactions remain uncharacterized due to a lack of high-throughput (HT) tools to study 3′UTR biology.
These possibly constitute a subset of many such interactions identified earlier that were shown, using miRNAs and genes of interest, to cause repression [ 12].
As described for filamin above, many such interactions are mediated by β-strand augmentation or complementation [ 56], whereby a single β-stranded peptide binds to the β-sheet of its partner, forming a mechanically long-lived complex.
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In many situations, such interactions may have limited, or at least rather subtle, effects.
However, despite much progress, the diverse ways in which these pathways interact to regulate cell fate and morphogenesis remain an area of intense research [ 1], and many more such interactions likely remain uncharacterized.
They reported that, while many studies show such interactions with HA experience of typically less than about 2 months ('short-term'), examinations considering long-term HA usage are still lacking.
Although many examples of such interactions can be cited, here we focus on the role of extracellular pH as a mechanism by which the environment affects the cancer cells and vice versa.
There have been many published studies exploring such interactions, particularly IGP, and to a lesser extent, competition (Phoofolo & Obrycki, 1998; Ware et al., 2009).
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