Sentence examples for many neural cells from inspiring English sources

Here we report GFP expression under the control of a putative promoter for F41D9.1 that occurs predominantly in many neural cells in the head around the posterior pharyngeal bulb, along the ventral nerve cord and in the tail (Fig 2).

In Platynereis, Pdu-sna1 is expressed in the mesoderm, but not in the foregut cells and only in a limited set of cells of the nervous system whereas Pdu-sna2 is expressed in many neural cells (including in the brain), in many foregut cells, but not in the mesoderm.

First, the use of a unique cellular model composed of two clones of PC12 cells: the wtPC12, with very low levels of REST, a property common to mature neurons and many neural cells, and the hrPC12, spontaneously expressing very high levels of the repressor.

First, in vertebrates, genes of the neurogenin family (ngn1, ngn2, and ngn3) are required for the formation of the precursors of many neural cells of both the Peripheral and Central Nervous Systems (PNS and CNS) [ 8- 10] while their single Drosophila ortholog, tap/biparous, has no proneural role and is expressed in a few differentiating neural cells [ 11, 12].

Connections between individual neurons are destroyed in the AD brain due to the formation of neurofibrillary tangles (i.e., tangled strands of neural fibers within the bodies of neurons) and senile plaques (i.e., masses of dying neural material containing toxic protein called beta-amyloid) which obstruct, damage and lead to the eventual death of many neural cells [ 1].

Cell surface marker expression has been described for the identification and isolation of many neural cell types by FACS from embryonic and adult tissue from multiple species.

Given the 50% reduction in ATP in Plp1tg brains, oxidative phosphorylation deficiencies in many neural cell types might be predicted.

Many neural cell adhesion and transmembrane molecules involved in signal transduction during neurite outgrowth and pathfinding contain immunoglobulin-like (Ig) motifs and localise to lipid rafts [ 249, 250].

On the one hand, many neural cell-specific proteins encoded by REST target genes, such as those of regulated neurosecretion, are lacking (D'Alessandro et al., 2008); on the other hand, PC12-27 cexhibithibit accelerated cell proliferation, controlled by a signaling loop in which REST operates together with the GAP protein TSC2 and the co-transcription factor β-catenin (Tomasoni et al., 2011).

While LPA and S1P have been reported to activate Akt in numerous systems including many neural cell types (Ye et al., 2002; Kim et al., 2008), Hla and colleagues have described opposing effects of S1P on Akt signaling in Human umbilical vein endothelial cells (HUVECs).

If too many neural progenitor cells exit the cell cycle during the early stages of development, then the overall number of cells is reduced, and the ratio of cell types shifts [ 1, 2].