Sentence examples for many human inflammatory from inspiring English sources

VAP-1 is upregulated in many human inflammatory diseases, such as acute myocardial infarction, inflammatory bowel disease and rheumatoid arthritis [45 48]; and a fully humanized anti-VAP-1 antibody is used in clinical trials for patients suffering from rheumatoid arthritis and psoriasis [49].

CXCL10 expression is strongly up-regulated in many human inflammatory diseases, including viral, bacterial and parasitic infections, skin diseases, atherosclerosis, allograft rejection, and others [1].

Additionally, MCs are considered to be critical effectors in many human inflammatory diseases [ 91, 92], and the core of different diseases including liver hepatitis [ 93], rheumatoid arthritis [ 94], arteriosclerosis [ 95], chronic graft-versus-host disease [ 96], and ischemic heart disease [ 97].

Clinical issue Tumor necrosis factor-α (TNFα) is a powerful pro-inflammatory cytokine that plays a prominent pathogenic role in many human diseases, including inflammatory and autoimmune disorders, ischemia-reperfusion injury and cancer.

Additionally, deregulated miRNA expression patterns have been documented in many human diseases including inflammatory and autoimmune diseases [ 7– 9].

It is overexpressed in many human cancers and inflammatory diseases; however, its exact role in inflammation is not known yet.

Alteration in the microbiota composition dysbiosis) results in immunological dysregulation that may underlie many human diseases such as inflammatory diseases [ 100], obesity [ 101], allergy [ 102] and autoimmunity [ 103].

These receptors are involved in many human diseases including chronic inflammatory diseases, degenerative diseases, autoimmune diseases, cancer, cardiovascular diseases etc, thus they could be of new therapeutic targets [ 24, 25].

Redox-sensitive transcription factors are often associated with the development and progression of many human disease states and inflammatory-related injury, particularly of the lung.

The reason for this is that redox-sensitive transcription factors are often associated with the development and progression of many human disease states and inflammatory-related injury, particularly of the lung [ 3].

Impaired beta-defensin synthesis has been described in many human diseased states, namely in inflammatory disorders [ 20, 22, 29, 30, 42- 44].