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These findings, that eater mutant larvae lack all or almost all sessile crystal cells while retaining many crystal cells in circulation, suggests that the absence of eater does not impair crystal cell differentiation.
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In fact, the crystal of 2-ME-SOD1, like many crystal structures of SOD1, also has unique crystallographic cell parameters and crystal packing.
Notch (upregulated BTT09545_1) is involved is involved in many signaling events in development including the specification of crystal cells in Drosophila melanogaster[ 60].
More recent liquid crystal displays (LCDs) rely on liquid crystal cells that realign incoming polarized light.
Embryo-derived types include crystal cells and plasmatocytes [1], [2].
Three hemocyte types occur: plasmatocytes, crystal cells, and lamellocytes.
Counting crystal cells using the BcF6-CFP label revealed that eater mutant larvae had no lack of crystal cells; they appeared to have twice as many circulating crystal cells as w control larvae (Fig. 4Gi).
Since eater mutants lack sessile crystal cells, we used two alternative methods to count crystal cells.
Hence, Hml+Lz+ cells are fully mature crystal cells or differentiating crystal cells while Hml+Lz− cells are plasmatocytes.
While sessile crystal cells appeared absent, crystal cells were still visible in circulation in eater mutant larvae (Fig. 4C,D).
The process of biomineralization has been widely studied for many crystal systems.
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