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Many DBPs show carcinogenic, mutagenic and teratogenic properties [122].
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Our results are also consistent with extensive quantitative genotoxicity data on DBPs showing that brominated DBPs are generally more genotoxic and carcinogenic than chlorinated DBPs (Plewa et al. 2008; Richardson et al. 2007).
Laboratory studies show that many DBPs are mutagenic or carcinogenic, but epidemiologic studies to date have revealed only a modest association between DBP exposure and cancer in humans.
Many DBPs were new and have not been reported previously in either swimming pool or drinking waters.
Toxicological studies indicated since the late 1980s that inhalation and dermal absorption for many DBPs were important exposure routes.
In addition, many DBPs we identified are new and have not been reported previously in pool waters.
The residuals of the blood concentrations and SBP and DBP showed a skewed distribution.
In particular, Rev-erbα and DBP showed the clearest shifts in the peak times of expression.
In contrast to SBP, DBP showed a statistically significant group × phase interaction (F 5,115) = 4.16, p < 0.005).
For males, long-term exposure to DBP showed an excess risk of colon cancer.
They found higher baseline SBP and DBP showed a significant risk of development of ESRD.
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