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The atypical manifestations are described.
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A series of histopathological manifestations were described in celiac patients and also in rhesus macaques with gluten-sensitive enteropathy – a newly established non-human primate model of gluten sensitivity [1], [3], [4], [5].
Clinical manifestations were described as being similar to those in adults.
Also, a positive association between vitamin D intake and atopic manifestations was described [ 22].
The degenerative manifestations were described blinded and standardised and expressed as 'max scores', 'sum scores' and 'present vs. not present'.
Subsequently its clinical manifestations were described by a scholar in Yale University in 1977, and named Lyme disease in 1980 [ 1].
Other neurological manifestations that are described in the literature are vertigo, dysarthria, ataxia, tremor, paraesthesias, migraine and cavernous sinus thrombosis [14].
The intravenous route of exposure has added a new complexity to the immunopathogenic picture and has resulted in novel, severe, and highly variable patterns of manifestation which are described in the clinical cases section of this article.
Interventions for other manifestations of autonomic neuropathy are described in the ADA statement on neuropathy (301).
Interventions for other manifestations of autonomic neuropathy are described in an ADA statement on neuropathy (335).
Interventions for other manifestations of autonomic neuropathy are described in the ADA statement on neuropathy (306).
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