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Efforts should be expanded to incentivize males to test for HIV prior to the onset of symptoms.
When CKO females were mated with WT males to test the recombination rates in the oocytes, 44% of the deletion (19 out of 43 analyzed embryos) was premeiotic, and 49% of the deletion (21 out of 43) was pre- or post-meiotic.
Otherwise, four endos /Df or Df/+; endos /+ females were crossed to three y w males to test their fertility.
We then mated these conditional knockout females with wild-type males to test for development of fertilized Ago2-depleted oocytes.
To allow for this, we shall also introduce parameters g 4: the proportion of −α/ααββT σσ XDEFXDEF females or −α/ααββT σσ XDEFY males to test positive in a OTOFT, and g 5 : the proportion of −α/−αββT σσ XDEFXDEF females or −α/−αββT σσXDEFY males to test positive in a OTOFT.
These female F1 progeny were then outcrossed to w males to test for fertility; a cross of w females to w males was used as the wild-type control.
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The eggs of each female parent were divided in two equal groups so that one half could be fertilized by a wild male and the other by a hatchery male to test for maternal effects (Fig. 2).
We also quantified male scent marking (as a measure of males' quality) to test whether differences in male quality affected the rate of multiple paternity.
To evaluate growth patterns of ambulatory males with Duchenne muscular dystrophy (DMD) treated with corticosteroids compared with ambulatory, steroid-naïve males with DMD and age-matched unaffected general-population males and to test associations between growth and steroid treatment patterns among treated males.
We developed four polymorphic microsatellite markers and used them to conduct parentage analyses on 21 nests collected during the breeding season to examine the rates of multiple mating by males and to test for evidence of alternative mating strategies.
We performed general discriminant analysis using Statistica v9.1 to predict ASD susceptibility in affected males, and to test for evidence of gene-gene interactions of DA-related genes and ASDs.
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