Sentence examples for malaria models from inspiring English sources

Immunization with genetically engineered, attenuated malaria parasites (GAP) that arrest during liver infection confers sterile protection in mouse malaria models.

Immune response against circumsporozoite protein (CSP) of Plasmodium berghei, a major surface protein on the sporozoite, confers protection in various murine malaria models.

Maximal immune response to LPCscys+ in rodent malaria models depends upon formation of cross-linkages to generate homopolymers, presenting challenges for vaccine production.

While the host parasite relationship in humans has been difficult to determine, the pliability of murine malaria models has enabled valuable contributions to the understanding of the pathogenesis of disease.

Our data establish this quantitative real-time PCR assay to be a fast and reproducible way of accurately assessing liver stage parasite burden and vaccine efficacy in rodent malaria models.

This paper addresses the effect of the choice of the incidence function for the occurrence of backward bifurcation in two malaria models, namely, malaria model with standard incidence rate and malaria model with nonlinear incidence rate.

The present study evaluates the biological relevance of several LPCs using a well characterized rodent malaria model.

The murine malaria model parasite Plasmodium berghei is the genetically most amendable Plasmodium species and allows full access to the entire life cycle in vivo.

We consider the dynamics of a mosquito-transmitted pathogen in a multi-patch Ross Macdonald malaria model with mobile human hosts, mobile vectors, and a heterogeneous environment.

Rigorous qualitative analyzes of the models show that the malaria model with standard incidence rate exhibits the phenomenon of backward bifurcation whenever a certain epidemiological threshold, known as the basic reproduction number, is less than unity.

For the malaria model with nonlinear incidence rate, it is shown that this phenomenon does not occur and the disease-free equilibrium of the model is globally-asymptotically stable whenever the reproduction number is less than unity.