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The molecular epidemiology of the influenza viruses circulating in Asia, including Taiwan, is already of wide international concern because strong travel makes the viruses easy to spread between Asia and other countries [ 9].
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The third mutation, E627K, makes the virus more likely to replicate.
This in part is what makes the virus such a mystery, and so catchy.
But infectivity should come at a high cost: Exposing the CD4-binding site makes the virus vulnerable to antibody attack.
What is more plausible, some researchers say, is a change that makes the virus less deadly, but also harder to get rid of.
"Using genetic engineering it may be possible to modify the virus so it does produce the V protein or alternatively put the gene from Hendra virus into Cedar virus and see if that makes the virus pathogenic".
Webster stressed that the receptor binding ability was not the only factor that makes the virus so worrying.
The bacteria migrates to the lungs, causing pneumonia, which then makes the virus transmissible to other people by coughing.
Check the animation above to see what makes the virus tick, and how, thankfully, it's going to take much more than a couple of CS majors and a case of Redbull to reverse-engineer this monster for personal use.
The M184V mutation in HIV-1 RT makes the virus resistant to 3TC and -FTC [34].
It is this potential that makes the virus escape from the host-immune system and resistant to anti-retroviral drugs.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com