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The third mutation, E627K, makes the virus more likely to replicate.
This in part is what makes the virus such a mystery, and so catchy.
But infectivity should come at a high cost: Exposing the CD4-binding site makes the virus vulnerable to antibody attack.
What is more plausible, some researchers say, is a change that makes the virus less deadly, but also harder to get rid of.
"Using genetic engineering it may be possible to modify the virus so it does produce the V protein or alternatively put the gene from Hendra virus into Cedar virus and see if that makes the virus pathogenic".
Webster stressed that the receptor binding ability was not the only factor that makes the virus so worrying.
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But what mutations would make the virus more easily transmissible?
Fouchier hoped to characterize the properties that make the virus so much deadlier than others.
The study identifies particular mutations that made the virus spread so easily.
The mutation slightly changed the shape of the spike, making the virus more transmissible.
Just a few mutations was all it took to make the virus go airborne.
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