α-Glucosidase inhibitors specifically target postprandial hyperglycemia and have low hypoglycemia risk, making them theoretically attractive for older patients.
This quality makes them theoretically applicable for substitution of even large bone defects that are characterized by osteogenic insufficiency.
MPCs can be found resident within a host of musculoskeletal and connective tissues, and the multipotential nature of MPCs makes them theoretically ideal candidates for repair of cartilage defects, especially those that also involve subchondral bone.
AChE enzyme inhibitors can upregulate AChE transcription which in certain contexts can have deleterious (noncatalytic) effects, making them theoretically harmful in ALS, whilst AChE antisense-oligonucleotides (mEN101), which downregulate AChE may be beneficial.
Furthermore, blood ACA levels may reach 30 - 500 mM following ethanol intoxication in certain populations such as Asians and African American who carry a mutated ALDH with low K m (12), making them theoretically ideal to examine the role of ACA.
Thus, triggering TLR7/8 stimulates the innate as well as the adaptive immune response; this unique property makes them, at least theoretically, superior to cytokines, which are conventionally given as single agent and either act on the innate or the adaptive arm of the immune system.
Additionally, in 2006 scientists announced that they had genetically engineered cattle lacking a necessary gene for prion production – thus theoretically making them immune to BSE, building on research indicating that mice lacking normally occurring prion protein are resistant to infection by scrapie prion protein.
For example the intronic regions are extracted from coding sequences during splicing, which theoretically makes them available to the Drosha enzyme.
Simple sugars and fat were tightly controlled, and the diets were isocaloric in this study, theoretically making them healthier compared with diet options in the free-living environment.
