Sentence examples for make the neurons from inspiring English sources

Since 1NMPP1 selectively inhibits TrkB F616A activation by deoxygedunin, we hypothesized that blockade of TrkB F616A signaling by 1NMPP1 in mice would make the neurons vulnerable to KA-provoked neuronal cell death.

Not only that, but other genetic tweaks make the neurons actually emit light when active, so the optrode can both influence and monitor the steering pathways.

We find that external input exerts profound and sometimes counterintuitive changes in the correlation among neurons: for example, a strong input can make the neurons almost independent.

It is used to take image prior into account to fastly and accurately predict the image details and to make the neurons work on the local image patches to avoid compromise to different image contents.

In particular, we show that a strong input can make the neurons almost independent, suggesting that functional connectivity does not depend only on the static anatomical connectivity, but also on the external inputs.

We have shown that a strong input can make the neurons almost independent, and this reveals a simple mechanism to achieve network decorrelation that adds to those, such as the balance of excitation and inhibition (e.g. [27, 55]) or the use of purely inhibitory feedback (e.g. [56]), that were recently proposed.

The strong T1 response would make the neuron appear to fire more for the right reward location.

They therefore allow for potassium flux that opposes depolarization, hence their activation will make the neuron less excitable.

To this end we propose a formal definition of History-Dependent Excitability (HDE) at any moment in time as the minimal synaptic strength <img src="http://journals.plos.org/plosone/article/asset?id=info?doi/10.1371/journal.pone.0015023.e010.PNG" class= inline-graphic"/> of an excitatory synaptic event that can make the neuron fire.

If the pulses are short, higher current intensities are required to make the neuron fire, and the first action potential is fired with a shorter delay after the pulse onset.

According to this explanation, sodium channels would reopen (repolarisation) within a shorter time frame after depolarisation under TRNS compared to non-TRNS and thereby make the neuron ready for repeated excitation.