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Conversely, the majority of spikes (∼80%) are generated uniformly in most neurons despite their different biophysical characteristics.
As we showed, the majority of spikes were emitted in the same locations of the stimulus, therefore, spike responses were consistent across neurons from different groups.
In conclusion, we propose that the regular patterns, which comprise the majority of spikes in PC SS trains, can control the amplitude of subsequent timing signals by modulating the amplitude of rebound spikes in downstream DCN neurons.
The majority of spikes in L4 excitatory neurons encode tactile information.
A) The vast majority of spikes, in many cases >90%, can be accounted for in terms of tactile behavior.
The majority of spikes (209, 77%) were observed during the high amplitude widespread slow waves, the remaining spikes occurring during the control segments (P ≈ 0).
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However, the majority of spike trains we analyzed showed a very slow convergence to Gaussianity.
However, we observed that the majority of the spikes emitted by the three types of neurons occur in the same locations of the input waveform.
As we have shown for the known cell types (I, II and III), the majority of their spikes were trivial in the sense that they reflected more the dynamics of the synaptic input than the biophysical character of the neuron.
However, such somatic spikelets were only observed in a small minority of cells (∼5%) in response to high-frequency bursts of PP stimuli (Jarsky et al., 2005), indicating that the vast majority of local spikes generated at the distal apical dendrites failed to propagate to CA1 soma, consistent with our CA1 results.
The majority of inter-spike intervals occurs at 80 s.
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