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Furthermore, major organs from the MoS2 ICG-treated MoS2 ICG-treatedted, including the heart, liver, kidney, lung, and spleen.
Tissue samples from all major organs from all animals were fixed in 4% buffered paraformaldehyde for 24 48 h prior to routine embedding in paraffin wax.
Histological sections of the major organs from naïve mice and W-LHIT treated mice were collected and analyzed.
Therefore, we determined TLR5 expression and the ability of flagellin to trigger prototypical innate immune responses and apoptosis in major organs from mice.
Various major organs from adult mice as well as whole embryos at different developmental stages (E) were used for the analysis.
We examined the relative expression and spatial distribution of SAα2,3-GalG 1-3 GalNAc SAα2,3-GalG 1-3 GalNAc SAα2,3-GalG 1-3 GalNAcfrom normandpoSAα2,6-Galpigs by binding with lectins Maackia amureceptorsglutinins (MAA II) and Sambucus nigra aggluthein (SNA) respectively.
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But we should also be clear that the colon is a major organ for the removal of toxins from the body.
Tissue samples of all major organs were collected from each monkey for histopathologic and immunohistochemical examination and were immersion-fixed in 10% neutral buffered formalin.
At 1 day postinjection, tumor tissues and major organs were collected from the mice.
Major organs were collected from the euthanized OVCAR8/ADR tumor bearing mice at 24 h post-PTT.
At the predetermined time points of 24, 48, and 72 h the group of Re‐C1P5‐injected mice and the group of Lu‐C1P5‐injected mice were killed, their tumors and major organs removed, blotted from blood, weighed, counted for radioactivity in a gamma counter, and percentage of injected dose per gram (ID/g) organ/tissue was calculated.
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