Because of its role as a major human pathogen, genetic factors responsible for its pathogenicity are of great interest.
Influenza A virus (IAV) is a major human pathogen with the potential to become pandemic.
This work offers a framework for further investigations into the interactions between genomic sequences and regulatory factors governing the complex transcriptional program of this major human pathogen.
The HIV-1 virus has relatively recently established itself as a major human pathogen, rapidly diversifying into a variety of phylogenetic subtypes or clades (A G) and recombinants in different populations.
Several genomes of this major human pathogen have been publicly available for almost 10 years, but comprehensive links between virulence or epidemicity and genome content of the bacterium are still missing.
Staphylococcus aureus (SA), is a major human pathogen causing wide range of clinical infections, which has been further complicated by drug resistance like methicillin resistant S. aureus (MRSA), vancomycin intermediate S. aureus (VISA)/vancomycin resistant S. aureus (VRSA), etc.
In this review, the adaptation of the major human pathogen Staphylococcus aureus to its host environment during infection will be discussed, particularly in the context of new sequencing technologies which have opened a gateway towards understanding of the molecular processes underlying those adaptations.
Staphylococcus aureus is a major human pathogen that causes a wide range of clinical infections.
The obligate intracellular bacterium Chlamydia trachomatis is a major human pathogen and a main cause of genital and ocular diseases.
Influenza A virus is a major human pathogen responsible for seasonal epidemics as well as pandemic outbreaks.
S. stercoralis the major human pathogen species is an intestinal nematode that has the ability to tolerate host immune attack and survive within the human small intestine for decades and infects at least 100 million people (Concha, Harrington Jr, & Rogers, 2005; Liu & Weller, 1993).
