In fact, jaundice was not documented in households where maize levels were the highest (48,000 ppb and 24,400 ppb), highlighting that factors associated with developing aflatoxicosis after aflatoxin exposure are not fully characterized.
In the feeding trial, there was no effect (P>0.05) of maize cob level on feed intake for both Large White and Mukota pigs.
Although higher levels of overall methylation have been reported in maize, the levels of asymmetric methylation are significantly lower than symmetric methylation (approximately 5% CHH compared with 86% CG and 74% CHG methylation).
Polynomial regression analysis was used to determine the relationship between maize cob levels with digestibility of nutrients, feed intake, average daily gain (ADG) and feed conversion ratio (FCR).
We found no significant difference in homegrown maize aflatoxin levels in 2005 compared with 2006.
Significant differences were found in the geometric mean (GM) of market maize aflatoxin levels between districts.
In contrast to maize, higher levels of CNV were identified in high-recombination regions in soybean and barley [ 18, 24].
Also, the association between the aflatoxicosis cases and market maize aflatoxin levels is ecologic and subject to ecologic fallacy.
Makueni and Kitui, the districts with the highest number of aflatoxicosis cases, also had the highest market maize aflatoxin levels.
For all 3 years combined (2005, 2006, and 2007), maize aflatoxin levels ranged from nondetectable to 48,000 ppb.
