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Even when cancer stem cells (CSCs) in myeloid leukemia have been strictly depicted as the cells responsible for tumor maintenance, the cellular origin of ALL is still a fundamental matter in question [ 7, 8].
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These proteins are involved in the maintenance of the cellular cytoskeleton.
Genome stability is crucial for the complete maintenance of the cellular pathways that govern the cell cycle.
In general, diseases are characterized by dysregulation in the transcriptional machinery, which is responsible for the maintenance of the cellular homeostasis[78].
The maintenance of the cellular epigenomic landscape, which depends on the status of the one-carbon metabolic pathway, is essential for normal central nervous system development and function.
Hence, organization and integrity of the cytoskeleton were affected, which is particularly important for the maintenance of the cellular architecture and intracellular transport processes, and essential for the functionality and survival of neural cells.
In particular, it has been observed that collagen type VI plays an essential role in cartilage development and the maintenance of the cellular microenvironment [10], [11].
Heat shock protein 90 (has90) has a key role in the maintenance of the cellular proteostasis.
This 30%% activity may be sufficient for the maintenance of the cellular ATP level.
NDPK is involved in the maintenance of the cellular NTP pool, transferring the phosphate group through the histidine phosphointermedier.
Moreover, IDH is essential for efficient glutathione recycling, and the maintenance of the cellular redox state [ 12, 13].
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com