Sentence examples for maintenance and plasticity from inspiring English sources

Contactin4 plays a crucial role in the formation, maintenance, and plasticity of neuronal networks.

In addition, these findings will contribute to our basic understanding of the cellular and molecular events involved in the formation, maintenance and plasticity of the neuromuscular synapse.

The brain-derived neurotrophic factor (BDNF /TrkB system plays an important role in the development, phenotypic maintenance and plasticity of specific neuronal populations.

Recent studies have revealed that AMPK also mediates the maintenance and plasticity of α-motoneurons, the neuromuscular junction, and skeletal muscle.

There is, however, no consensus as to its precise function in the nervous system, and it remains unclear or untested as to what its role is in the development, maintenance and plasticity of neuronal connectivity in the intact brain and whether it is essential for any of the purported functions which have been attributed to it based largely on in vitro bioassays.

Neuronal synapse formation, maintenance, and plasticity involve drastic changes in the composition of synaptic proteins, and the UPS plays an essential role in controlling local protein turnover during these processes (Ehlers, 2003; Bingol and Sheng, 2011).

After completion of cerebellar morphogenesis, Bergmann glia remain important for structural support [12], [13], but also serve additional roles in synapse maintenance, function and plasticity [14], [15], [16], [17].

In the adult retina and cerebellum, gain or loss of Norrin/Fz4 signaling results in a cell-autonomous gain or loss, respectively, of blood retina barrier and blood brain barrier function, indicating an ongoing requirement for Frizzled signaling in barrier maintenance and substantial plasticity in mature CNS vascular structure.

Here we examined synaptogenesis, synaptic maintenance and synaptic plasticity at cortical and sub-cortical excitatory projections to the LA in a model of RTT, Mecp2308/Y (KO) mice [16] and their Mecp2X/Y (WT) littermates.

To date the role of Mecp2 in the key phases of synaptic life – synaptogenesis, synaptic maintenance and synaptic plasticity - is still conflicting: A dramatic loss of excitatory synapses is observed in Mecp2-deficient primary neuronal cultures, but synaptic marker density appears to be normal in hippocampal slices of symptomatic RTT mice [3].

BDNF is the most abundant neurotrophin in the brain, playing a critical role in growth, differentiation, maintenance, and synaptic plasticity.