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ARC has a role in maintaining physiological cardiac function.
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In reasonable amounts it is a valuable stimulus to maintain physiological vigour.
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The ventilation settings can then be adjusted to try to maintain physiological levels of O2/CO2.
Short-term Akt activation in inducible Akt1 transgenic mice induces physiological cardiac hypertrophy with maintained vascular density [ 23], indicating that coronary angiogenesis is enhanced to keep pace with the growth of the myocardium.
Building on the Neo-Hookean model, the Neo-fiber law maintains the good identifiability characteristics, while reproducing physiological cardiac deformation.
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