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This may occur by controlling excitotoxicity and maintaining cellular adenosine 5′-triphosphate levels.
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As major energy-generating organelles in eukaryotic cells, mitochondria are essential in maintaining cellular energy supplies by generating adenosine triphosphate (ATP) through oxidative phosphorylation (OXPHOS).
Protein protein interactions (PPIs) are vital to maintaining cellular homeostasis.
Aquaporins (AQPs) are primarily involved in maintaining cellular water homeostasis.
Autophagy is of great significance in maintaining cellular homeostasis.
GRP94 plays an important role in maintaining cellular homeostasis.
The CWI pathway is essential for maintaining cellular integrity.
This leads to augmented myocardial adenosine triphosphate provision and maintains cellular energy charge during coronary ischemia, resulting in better tolerance to ischemia and improved myocardial protection [ 60].
Although the majority of the adenosine triphosphate (ATP) in chondrocytes is made by glycolysis rather than by oxidative phosphorylation in mitochondria there is evidence to suggest that reactive oxygen species produced by mitochondrial electron transport (ET) help to maintain cellular redox balance in favor of glycolysis.
HCA also maintained cellular viability, preventing stretch-induced cell death.
A glycolytic enzyme maintains cellular redox homeostasis during metabolic stress.
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